JITENDRA PATEL’s Post

Aging myocardium has a decreased chronotropic response to catecholamines and is dependent on preload (intravascular volume). Hypovolemia can easily result in shock. Deterioration of the cardiac conduction system leads to atrial fibrillation and bundle branch blocks.Medications, especially digoxin, -blockers, and calcium channel block-ers, impair the tachycardic response to catecholamines, both impairing the body's inability to compensate for hemorrhage and making heart rate an unreliable predictor of hypovolemia.

Hyperkalemia: A condition characterized by elevated potassium levels in the blood, typically >5.5 mmol/L. It can lead to life-threatening cardiac arrhythmias ECG Changes Based on Potassium Levels 1. Mild Hyperkalemia (5.5–6.0 mmol/L): Peaked T waves (narrow and tall). 2. Moderate Hyperkalemia (6.1–6.9 mmol/L): Prolonged PR interval. Flattening or loss of P waves. Widened QRS complex. 3. Severe Hyperkalemia (>7.0 mmol/L): Sine wave pattern (merging of QRS and T waves). Ventricular fibrillation or asystole (cardiac arrest).

Did you know that iodine plays a crucial role in nitric oxide production? Nitric oxide is a vital molecule that supports cardiovascular health, blood flow, and overall well-being. Learn how to ensure adequate iodine intake and reap the benefits of enhanced nitric oxide production. HOW TO ACCESS THE EPISODE 📹 Watch the episode in full https://lnkd.in/gyp7NJKC

Comprehensive non-invasive Hemodynamic Assessment in Acute Decompensated Heart Failure-Related Cardiogenic Shock. A step towards echodynamics: @ESC_Journals 🥸Nice study looking at correlations between echo and RHC in CS 👇👇👇 https://lnkd.in/e8UpcfFp

Safety and efficacy of 2% vs 3% Peripheral Hypertonic Sodium Chloride Administration in patients with severe hyponatremia, elevated intracranial pressure or cerebral edema. https://lnkd.in/guCkk4sX The figure below shows the response of sodium levels to the infusion.

Surprised to see single coronary artery, anomalous originating from right coronary sinus, giving rise to all the three coronary arteries.. presented with acute coronary syndrome, very critical stenosis of left circumflex artery… performed PCI using Biomime morph 3 -2.5 mm x 60 mm successfully. Identify the vessels…. #meryllifesciences #biomimemorph

#Mitral annular calcification (MAC) is a chronic degenerative process characterized by calcium deposits in the mitral valve (MV) apparatus, most commonly affecting the central posterior MV annulus Often an incidental imaging finding, MAC is associated with cardiovascular morbidity & mortality bit.ly/3YrSnXU

Non-Invasive Early Warning System of Systemic Hypoperfusion: Circulatory Shock and Sepsis - Part I: The Problem   by Michael R. Pinsky, MD and Jacques Creteur, MD   The Problem:  Circulatory shock is defined as an inadequate oxygen (O2) delivery to tissue to sustain metabolic demand. If arterial oxygen content is adequate, then tissue ischemia develops only at the very extremes of low blood flow. Well before that time, normal physiologic adaptive mechanisms controlled by the autonomic nervous system and mediated primarily through increased sympathetic tone tend to sustain an adequate central arterial blood pressure despite falling total blood flow. Once this regulatory process is exhausted, however, systemic hypotension develops. Thus, systemic hypotension, defined as a mean arterial pressure <65 mmHg or a systolic arterial pressure <90 mmHg, occurs late in shock when tissue hypoperfusion is already compromising metabolic function. If circulatory shock associated with systemic hypotension persists, then generalized tissue ischemia manifests as end-organ failure, lactic acidosis and autonomic failure. If the bedside clinician waits for systemic hypotension to recognize circulatory insufficiency before treating their patient for circulatory shock, then he will have waited too long. #shock #hypoperfusion #sepsis #circulation

Basics of ECG part 59 First question: Consider the blockage in a coronary artery, what will happen now? 1)If the blockage is incomplete ,then the lack of oxygen to the cardiac tissue is named “Ischemia”.Note that the ischemia also may be due to myocardial oxygen supply/demand mismatch. 2)If the blockage is complete ,the situation is termed ”Injury “,it means that there is reversible myocardial damage due the artery obstruction and the blockage of cell supply (oxygen,glucose,……). 3)If the blockage is complete and persistent,this condition leads to irreversible myocardial damage,so it’s called ”Infarction “. So ,you see the ischemia, injury, and infarction of cardiac tissue are the three consecutive situation. Now,the second question: What are the changes in the ECG of each of these three situations ? We will discuss them in the next part. #ECG #cardilogy

https://lnkd.in/diui5JTZ To remember the H's and T's in the context of Advanced Cardiac Life Support (ACLS), you can use this mnemonic song set to the tune of "Twinkle, Twinkle, Little Star": **"Hypovolemia, lack of flow, Hypoxia, oxygen's low. Hyper or hypokalemia, watch for these, Hypothermia, freezing breeze.** **Tension pneumo, chest so tight, Tamponade, heart's in a fight. Thrombosis, clots block the way, Toxins, trouble every day.** **Hypoglycemia, sugar's fall, These H’s and T’s, we recall."**