JITENDRA PATEL’s Post

Understanding Hyperkalemia and Its Dangers Hyperkalemia is a potentially life-threatening condition characterized by elevated serum potassium levels (>5.5 mmol/L). It disrupts cardiac conduction, leading to arrhythmias. ECG changes are progressive: mild hyperkalemia (5.5–6.0 mmol/L) shows peaked T waves; moderate hyperkalemia (6.1–6.9 mmol/L) may result in prolonged PR intervals, flattened P waves, and widened QRS complexes. Severe hyperkalemia (>7.0 mmol/L) can cause a sine wave pattern, ventricular fibrillation, or asystole. Prompt management includes calcium gluconate for cardioprotection, insulin with glucose to shift potassium intracellularly, and measures to eliminate potassium, such as diuretics or dialysis. Early recognition saves lives.

very well summarised and demonstrated in one short video

Hyperkalemia: A condition characterized by elevated potassium levels in the blood, typically >5.5 mmol/L. It can lead to life-threatening cardiac arrhythmias ECG Changes Based on Potassium Levels 1. Mild Hyperkalemia (5.5–6.0 mmol/L): Peaked T waves (narrow and tall). 2. Moderate Hyperkalemia (6.1–6.9 mmol/L): Prolonged PR interval. Flattening or loss of P waves. Widened QRS complex. 3. Severe Hyperkalemia (>7.0 mmol/L): Sine wave pattern (merging of QRS and T waves). Ventricular fibrillation or asystole (cardiac arrest).

Comprehensive non-invasive Hemodynamic Assessment in Acute Decompensated Heart Failure-Related Cardiogenic Shock. A step towards echodynamics: @ESC_Journals 🥸Nice study looking at correlations between echo and RHC in CS 👇👇👇 https://lnkd.in/e8UpcfFp

Did you know that iodine plays a crucial role in nitric oxide production? Nitric oxide is a vital molecule that supports cardiovascular health, blood flow, and overall well-being. Learn how to ensure adequate iodine intake and reap the benefits of enhanced nitric oxide production. HOW TO ACCESS THE EPISODE 📹 Watch the episode in full https://lnkd.in/gyp7NJKC

#Mitral annular calcification (MAC) is a chronic degenerative process characterized by calcium deposits in the mitral valve (MV) apparatus, most commonly affecting the central posterior MV annulus Often an incidental imaging finding, MAC is associated with cardiovascular morbidity & mortality bit.ly/3YrSnXU

Functional assessment in angina and non-obstructive coronary arteries: from microvascular resistance reserve to subtypes of coronary microvascular dysfunction https://lnkd.in/duTvM_bs

Are you interested in functional assessment in angina and non-obstructive coronary arteries (ANOCA) patients? 📢Read our paper, just published in the Journal of Cardiovascular Medicine! Key findings 💡: ✔️ MRR and CFR relationship 📈 ✔️ The role of IMR in different CMD subtypes 🔍 ✔️ Clinical and functional characteristics of different CMD subtypes 🩺📊 ✔️ The importance of #FullPhysiology! A special thank to my mentor Prof. Antonio Maria Leone

Surprised to see single coronary artery, anomalous originating from right coronary sinus, giving rise to all the three coronary arteries.. presented with acute coronary syndrome, very critical stenosis of left circumflex artery… performed PCI using Biomime morph 3 -2.5 mm x 60 mm successfully. Identify the vessels…. #meryllifesciences #biomimemorph

Non-Invasive Early Warning System of Systemic Hypoperfusion: Circulatory Shock and Sepsis - Part I: The Problem   by Michael R. Pinsky, MD and Jacques Creteur, MD   The Problem:  Circulatory shock is defined as an inadequate oxygen (O2) delivery to tissue to sustain metabolic demand. If arterial oxygen content is adequate, then tissue ischemia develops only at the very extremes of low blood flow. Well before that time, normal physiologic adaptive mechanisms controlled by the autonomic nervous system and mediated primarily through increased sympathetic tone tend to sustain an adequate central arterial blood pressure despite falling total blood flow. Once this regulatory process is exhausted, however, systemic hypotension develops. Thus, systemic hypotension, defined as a mean arterial pressure <65 mmHg or a systolic arterial pressure <90 mmHg, occurs late in shock when tissue hypoperfusion is already compromising metabolic function. If circulatory shock associated with systemic hypotension persists, then generalized tissue ischemia manifests as end-organ failure, lactic acidosis and autonomic failure. If the bedside clinician waits for systemic hypotension to recognize circulatory insufficiency before treating their patient for circulatory shock, then he will have waited too long. #shock #hypoperfusion #sepsis #circulation